Treatment of Stanford Type A dissection with E-vita Open stent graft, a life-saving surgery

    Authors

    Keywords

    aortic dissection, cerebral infarction, E-vita stent graft

    DOI

    https://doi.org/10.15836/ccar2022.290

    Full Text

    Introduction: Aortic dissection is a high mortality rate disease with incidence of 2,5-3,5/100000 people per year ( 1 , 2 ). Primary manifestation of aortic dissection is sudden and persistent chest and back pain. 1/3-1/2 aortic dissection with neurological symptoms have no chest pain. Without surgical intervention, dissection mortality at 3 days after onset of symptoms is greater than 50% ( 3 ). Case report : 58-year-old female previously healthy, initially presented with cerebrovascular insult and right sided hemiparesis. On admission, patient was unconscious, without verbal contact, anisocoria was presented. Head Computed tomography (CT) scan did not show signs of ischemia, hemorrhage, or tumor. CT angiography showed dissection of left internal carotid artery and verified diagnosis of acute aortic dissection Stanford type A with retrograde intramural hematoma and large pericardial effusion ( Figure 1 ) . Due to threatening tamponade, pericardiocentesis was performed, and guide wire was place in the true lumen through femoral artery. Patient was immediately transferred to the operating room. The replacement of the root and ascending aorta graph with reconstruction of coronary arteries was performed (sec Bentall). Aortic arch was replaced with reimplantation of supra-aortic branches and implantation of stent graft in thoracic aorta (Evita Open Neo) ( Figure 2 ) . Circulatory arrest lasted 36 minutes, operation was finished without complications. She was extubated on first postoperative day, with significant neurological improvement fourth day. Control CT aortography showed proper flow through graft, coronary arteries, supra-aortal branches, with no signs of paravalvular endoleak or pseudoaneurysm. Head CT scan showed hypodense areas in right hemisphere in terms of acute embolic ischemia. Echocardiography showed good function of mechanical aortic valve (mean pressure gradient 10 mmHg, aortic valve area velocity time integral 2,2 cm 2 ), without regurgitation and normal ejection fraction of the left ventricle. Intensified physical therapy led to a complete neurological recovery. Patient was discharged nineteenth postoperative day in good condition. CT angiography image shows dissection of the ascending aorta and intramural hematoma. Intraoperative image of replaced aortic arch with reimplantation of supra-aortic branches and implantation of a stent graft in the thoracic aortae (Evita Open Neo 26/24). Conclusion: Considering the atypical manifestation of aortic dissection in forms of neurological symptoms, such patients represent a demanding challenge in establishing the diagnosis as well as in its prompt treatment.

    Cardiologia Croatica
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    Treatment of Stanford Type A dissection with E-vita Open stent graft, a life-saving surgery

    Extended Abstract
    Issue9-10
    Published
    Pages290
    PDF via DOIhttps://doi.org/10.15836/ccar2022.290
    aortic dissection
    cerebral infarction
    E-vita stent graft

    Authors

    Dubravka Šušnjar*ORCIDDubrava University Hospital, Zagreb, Croatia
    Josip VarvodićORCIDDubrava University Hospital, Zagreb, Croatia
    Savica GjeorgjievskaORCIDDubrava University Hospital, Zagreb, Croatia
    Nikola SliškovićORCIDDubrava University Hospital, Zagreb, Croatia
    Igor RudežORCIDDubrava University Hospital, Zagreb, Croatia

    Full Text

    Introduction: Aortic dissection is a high mortality rate disease with incidence of 2,5-3,5/100000 people per year ( 1 , 2 ). Primary manifestation of aortic dissection is sudden and persistent chest and back pain. 1/3-1/2 aortic dissection with neurological symptoms have no chest pain. Without surgical intervention, dissection mortality at 3 days after onset of symptoms is greater than 50% ( 3 ). Case report : 58-year-old female previously healthy, initially presented with cerebrovascular insult and right sided hemiparesis. On admission, patient was unconscious, without verbal contact, anisocoria was presented. Head Computed tomography (CT) scan did not show signs of ischemia, hemorrhage, or tumor. CT angiography showed dissection of left internal carotid artery and verified diagnosis of acute aortic dissection Stanford type A with retrograde intramural hematoma and large pericardial effusion ( Figure 1 ) . Due to threatening tamponade, pericardiocentesis was performed, and guide wire was place in the true lumen through femoral artery. Patient was immediately transferred to the operating room. The replacement of the root and ascending aorta graph with reconstruction of coronary arteries was performed (sec Bentall). Aortic arch was replaced with reimplantation of supra-aortic branches and implantation of stent graft in thoracic aorta (Evita Open Neo) ( Figure 2 ) . Circulatory arrest lasted 36 minutes, operation was finished without complications. She was extubated on first postoperative day, with significant neurological improvement fourth day. Control CT aortography showed proper flow through graft, coronary arteries, supra-aortal branches, with no signs of paravalvular endoleak or pseudoaneurysm. Head CT scan showed hypodense areas in right hemisphere in terms of acute embolic ischemia. Echocardiography showed good function of mechanical aortic valve (mean pressure gradient 10 mmHg, aortic valve area velocity time integral 2,2 cm 2 ), without regurgitation and normal ejection fraction of the left ventricle. Intensified physical therapy led to a complete neurological recovery. Patient was discharged nineteenth postoperative day in good condition. CT angiography image shows dissection of the ascending aorta and intramural hematoma. Intraoperative image of replaced aortic arch with reimplantation of supra-aortic branches and implantation of a stent graft in the thoracic aortae (Evita Open Neo 26/24). Conclusion: Considering the atypical manifestation of aortic dissection in forms of neurological symptoms, such patients represent a demanding challenge in establishing the diagnosis as well as in its prompt treatment.