Authors
- Josip Silović — General Hospital “Dr. Josip Benčević”, Slavonski Brod, Croatia — ORCID: 0009-0002-9918-7575
- Domagoj Mišković — General Hospital “Dr. Josip Benčević”, Slavonski Brod, Croatia — ORCID: 0000-0003-4600-0498
- Ivan Majdandžić — General Hospital “Dr. Josip Benčević”, Slavonski Brod, Croatia — ORCID: 0009-0006-0014-6642
- Marijana Knežević Praveček — General Hospital “Dr. Josip Benčević”, Slavonski Brod, Croatia — ORCID: 0000-0002-8727-7357
- Katica Cvitkušić Lukenda — General Hospital “Dr. Josip Benčević”, Slavonski Brod, Croatia — ORCID: 0000-0001-6188-0708
Keywords
coronary care unit, sedation, propofol, dexmedetomidine, remifentanil
DOI
https://doi.org/10.15836/ccar2026.46Full Text
**Introduction**: Modern cardiac catheterization laboratories and coronary care units (CCU) require sophisticated sedation protocols that balance patient comfort with hemodynamic stability and respiratory safety (1). This review presents sedation options currently implemented at General Hospital Slavonski Brod’s CCU. **Protocols and Methods**: We analyzed seven primary sedation agents used in our institution: propofol and propofol TCI (Target-Controlled Infusion), remifentanil TCI, dexmedetomidine, sufentanil, fentanyl, and midazolam. Each agent’s pharmacokinetics, pharmacodynamics, indications, contraindications, and adverse effects were systematically reviewed based on current literature and institutional experience (**Table 1** and **Table 2****).** Our sedation protocols are tailored to specific procedures: propofol for electrical cardioversion due to rapid onset and recovery (2); midazolam combined with propofol for transcatheter aortic valve implantation procedures; dexmedetomidine for mechanically ventilated patients post-cardiac arrest or myocardial infarction; and dexmedetomidine-based protocols for anxious elderly patients and non-invasive ventilation (NIV) mask tolerance enhancement (3, 4). Each approach considers patient-specific factors including age, comorbidities, and procedural complexity. Propofol demonstrates rapid onset (30-40 seconds) with significant cardiovascular depression but predictable recovery. TCI systems provide superior concentration control and reduced side effects. Remifentanil TCI offers precise analgesia control with ultra-short elimination half-life (3-10 minutes) but requires vigilant respiratory monitoring. Dexmedetomidine provides unique conscious sedation with minimal respiratory depression, making it ideal for prolonged sedation and NIV tolerance (3, 4). Traditional opioids (fentanyl, sufentanil) and midazolam remain valuable for specific indications with established safety profiles. Each agent presents distinct contraindication patterns. Propofol requires caution in cardiac failure and hypovolemia (2). Dexmedetomidine necessitates monitoring for bradycardia and hypotension. Opioids demand respiratory surveillance, while midazolam may cause paradoxical reactions in elderly patients. ### TABLE 1: Contraindications and special warnings. | **Drug** | **Absolute contraindications** | **Relative contraindications** | **Special warnings** | | --- | --- | --- | --- | | Propofol | Allergy to propofol, eggs, soy | Heart failure, hypovolemia | Propofol infusion syndrome, hypertriglyceridemia | | Remifentanil TCI | Allergy to fentanyl analogues | Severe renal/hepatic insufficiency, COPD, mechanical obstruction of the GIT | Muscle rigidity, respiratory depression | | Dexmedetomidine | No absolute contraindications | Bradycardia <50/min, hypotension, heart block | Bradycardia, prolonged action in the elderly | | Sufentanil/ Fentanyl | Allergy to opioid analgesics, acute asthma | CNS depression, increased intracranial pressure | Respiratory depression, addiction | | Midazolam | Allergy to benzodiazepines, acute glaucoma | Dementia, COPD, myasthenia gravis | Delirium in the elderly, anterograde amnesia | [†] TCI – Target-Controlled Infusion; COPD – Chronic Obstructive Pulmonary Disease; CNS – Central Nervous System; GIT – Gastrointestinal Tract ### TABLE 2: Pharmacokinetics and pharmacodynamics. | Drug | **Mechanism of action** | **Onset of action** | **Elimination half-time** | **Cardiovascular effects** | **Respiratory effects** | | --- | --- | --- | --- | --- | --- | | Propofol | GABA receptor agonist | 30-40 seconds | 4-7 hours | Hypotension, ↓CO, bradycardia | Respiratory depression, apnea | | Propofol TCI | GABA receptor agonist | 30-40 seconds | 4-7 hours | Hypotension, ↓CO, bradycardia | Respiratory depression, apnea | | Remifentanil TCI | μ-opioid receptor agonist | 1-3 minutes | 3-10 minutes | Bradycardia, hypotension | Significant respiratory depression | | Dexmedetomidine | α2-adrenoreceptor agonist | 15 minutes (without loading dose) | 2-3 hours | Bradycardia, hypotension, initial hypertension | Minimal respiratory depression | | Sufentanil | μ-opioid receptor agonist | 1-3 minutes | 2.5-3 hours | Bradycardia, mild hypotension | Significant respiratory depression | | Fentanyl | μ-opioid receptor agonist | 1-2 minutes | 3-4 hours | Bradycardia, mild hypotension | Moderate respiratory depression | | Midazolam | GABA-A receptor agonist | 1-3 minutes | 1-4 hours | Minimal | Mild respiratory depression | [†] TCI – Target-Controlled Infusion; ↓CO – Decreased Cardiac Output; GABA – gamma-aminobutyric acid **Conclusions**: Successful sedation in cardiac catheterization laboratories and coronary units require individualized approaches based on pharmacological understanding, procedural requirements, and patient characteristics. Our institutional experience demonstrates that combined protocols utilizing multiple agents can optimize patient outcomes while maintaining safety. Continuous monitoring and staff education remain paramount for safe sedation practice.
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