Authors
- Antonio Hanžek — University Hospital Centre Zagreb, Zagreb, Croatia — ORCID: 0000-0003-2308-3518
- Zvonimir Ostojić — University Hospital Centre Zagreb, Zagreb, Croatia — ORCID: 0000-0003-1762-9270
- Filip Lončarić — University Hospital Centre Zagreb, Zagreb, Croatia — ORCID: 0000-0002-7865-1108
- Luka Perčin — University Hospital Centre Zagreb, Zagreb, Croatia — ORCID: 0000-0003-0497-6871
- Tomislav Krčmar — University Hospital Centre Zagreb, Zagreb, Croatia — ORCID: 0000-0003-4689-1673
- Kristina Marić-Bešić — University Hospital Centre Zagreb, Zagreb, Croatia — ORCID: 0000-0002-4004-7271
- Davor Radić — University Hospital Centre Zagreb, Zagreb, Croatia — ORCID: 0000-0002-9132-1568
- Marijan Pašalić — University Hospital Centre Zagreb, Zagreb, Croatia — ORCID: 0000-0002-3197-2190
- Denis Došen — University Hospital Centre Zagreb, Zagreb, Croatia — ORCID: 0000-0003-3490-5505
- Hrvoje Jurin — University Hospital Centre Zagreb, Zagreb, Croatia — ORCID: 0000-0002-2599-553X
- Boško Skorić — University Hospital Centre Zagreb, Zagreb, Croatia — ORCID: 0000-0001-5979-2346
- Eduard Margetić — University Hospital Centre Zagreb, Zagreb, Croatia — ORCID: 0000-0001-9224-363X
- Davor Miličić — University Hospital Centre Zagreb, Zagreb, Croatia — ORCID: 0000-0001-9101-1570
- Joško Bulum — University Hospital Centre Zagreb, Zagreb, Croatia — ORCID: 0000-0002-1482-6503
Abstract
**Introduction**: The efficiency of drug-coated balloon (DCB) percutaneous coronary intervention (PCI) has been shown for in-stent restenosis (ISR) and native small-vessel disease, with available data showing similar outcomes in both chronic kidney disease (CKD) and non-CKD patients (1). The aim is to compare the incidence of target lesion restenosis at follow-up (FUP) coronary angiography in patients with and without CKD receiving DCB PCI. **Patients and Methods**: The registry included patients undergoing a DCB PCI at the University Hospital Centre Zagreb from February 2011 to January 2023 (n=652). Patient demographics, comorbidities, pharmacotherapy, as well as data on the initial and FUP coronary angiography/PCI was collected. Chronic kidney disease was defined as estimated glomerular filtration rate < 45 ml/min/1.73m2. A FUP angiography was performed in 49% of patients (n=317), with a median FUP of 6 (interquartile range 3 - 18) months, without difference between groups. **Results**: Data is shown in **Table 1**. The cohort was 75% male, mean age 65 ± 10 years. CKD was present in 9% (n=57) of patients and was associated with a higher incidence of arterial hypertension, diabetes mellitus, atrial fibrillation, as well as peripheral artery disease. The age difference was noted between groups, with CKD patients being older on average. At initial PCI, more CKD patients had multivessel coronary disease, with a higher rate of ISR as the indication for DCB, that was not statistically significant (CKD vs non-CKD: 46% vs 34%, p=0.075). After DCB, no difference was noted between groups in regards to the need for a bail-out PCI (9% VS 6%, P=0.375). FUP was performed in an equal percentage of patients in both groups (48% vs 51%, p=0.769), with no differences seen in the incidence of restenosis (17% vs. 18%, p=0.998), the need for target lesion PCI (17% vs. 13%, p=0.533), or the use of anti-anginal drugs. ### TABLE 1: Comparison between chronic kidney disease (CKD) and non-CKD patients. | | | Patients with chronic kidney disease (n=57) | Patients without chronic kidney disease (n=585) | P -value | | --- | --- | --- | --- | --- | | Initial PCI hospitalization | | | | | | Age, years (mean ± SD) | | 72 ± 9 | 64 ± 10 | <0.001* | | Male sex, n (%) | | 38 (67) | 445 (76) | 0.147 | | History of myocardial infarction, n (%) | | 31 (54) | 246 (42) | 0.073 | | History of PCI, n (%) | | 39 (68) | 347 (59) | 0.180 | | History of CABG, n (%) | | 5 (9) | 21 (4) | 0.071 | | History of stroke or TIA, n (%) | | 7 (12) | 34 (6) | 0.057 | | History of atrial fibrillation, n (%) | | 19 (33) | 61 (10) | <0.001* | | History of peripheral artery disease, n (%) | | 11 (19) | 44 (8) | <0.002* | | Arterial hypertension, n (%) | | 55 (97) | 502 (86) | 0.022* | | Diabetes mellitus, n (%) | | 32 (56) | 190 (33) | <0.001* | | ACS as indication for DCB PCI, n (%) | | 25 (44) | 275 (47) | 0.679 | | Multivessel coronary disease, n (%) | | 39 (70) | 292 (50) | 0.013* | | In-stent restenosis, n (%) | | 25 (46) | 194 (34) | 0.075 | | Bail-out PCI, n (%) | | 5 (9) | 35 (6) | 0.375 | | Follow-up hospitalization | | | | | | Elective procedure, n (%) | | 18 (75) | 244 (84) | 0.261 | | Restenosis of target DCB PCI lesion, n (%) | FUP cohort (n= 317) | 4 (17) | 51 (18) | 0.998 | | Whole cohort (n=645) | 4 (7) | 51 (9) | 0.808 | | [†] SD – standard deviation, PCI – percutaneous coronary intervention, CABG – coronary artery bypass graft, TIA – transient ischemic attack, ACS – acute coronary syndrome, DCB – drug-coated balloon *p<0.05 **Conclusion**: The findings of our single-centre analysis show that patients with CKD do not have a higher risk of target lesion restenosis after DCB PCI, when compared to the non-CKD group, which is in accordance with currently available evidence (2).
Keywords
chronic coronary disease, drug-coated balloons, PCI, restenosis, chronic kidney disease
DOI
https://doi.org/10.15836/ccar2023.70Literature
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