Hyperthyroidism caused by heart failure treatment

    Authors

    Keywords

    heart failure, hyperthyreoidism, nurses care

    DOI

    https://doi.org/10.15836/ccar2016.571

    Full Text

    Heart failure is a complex clinical syndrome that regardless of the diseases’ etiology is characterized by inability of the heart to receive and pump the blood thus meeting the metabolic needs of the body. Heart failure treatment often requires the introduction of amiodarone into therapy, which can, in some patients, cause the hyper production of thyroid gland hormones. (1, 2) Thyroid gland controls the metabolism through hormones (the way the body uses energy, breathing, heart rhythm, systemic nervous system, body weight, body temperature and many other functions in the body). Therefore, the occurrence of hyperthyroidism in patients with heart failure leads to progression of heart failure and further deterioration of patient health. Nurses’ care for such patients is more complex and involves timely implementation of therapy, daily physical examination, control of vital functions, keeping the balance of fluids, weight control, telemetric monitoring, psychological support. It is of utmost importance to recognize the changes in any of the mentioned segments so therefore enabling timely intervention and in doing so helping the patient to achieve the regression of hyperthyroidism and thus ‘stabilization’ of heart failure.

    Literature

    1. Narayana SK, Woods DR, Boos CJ. Management of amiodarone-related thyroid problems. Ther Adv Endocrinol Metab. 2011;2(3):115–26. https://doi.org/10.1177/2042018811398516
    2. Harjai KJ, Licata AA. Amiodarone-induced hyperthyroidism: a case series and brief review of literature. Pacing Clin Electrophysiol. 1996;19(11 Pt 1):1548–54. https://doi.org/10.1111/j.1540-8159.1996.tb03179.x
    Cardiologia Croatica
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    Hyperthyroidism caused by heart failure treatment

    Extended Abstract
    Issue10-11
    Published
    Pages571
    PDF via DOIhttps://doi.org/10.15836/ccar2016.571
    heart failure
    hyperthyreoidism
    nurses care

    Authors

    Lidija Ban*ORCIDUniversity Hospital Centre “Sestre milosrdnice”, Zagreb, Croatia
    Ivana TomašićORCIDUniversity Hospital Centre “Sestre milosrdnice”, Zagreb, Croatia
    Božica LeškoORCIDUniversity Hospital Centre “Sestre milosrdnice”, Zagreb, Croatia
    Vesna SlonjšakORCIDUniversity Hospital Centre “Sestre milosrdnice”, Zagreb, Croatia

    *Correspondence email: lidijaban2@gmail.com

    Full Text

    Heart failure is a complex clinical syndrome that regardless of the diseases’ etiology is characterized by inability of the heart to receive and pump the blood thus meeting the metabolic needs of the body. Heart failure treatment often requires the introduction of amiodarone into therapy, which can, in some patients, cause the hyper production of thyroid gland hormones. (1, 2) Thyroid gland controls the metabolism through hormones (the way the body uses energy, breathing, heart rhythm, systemic nervous system, body weight, body temperature and many other functions in the body). Therefore, the occurrence of hyperthyroidism in patients with heart failure leads to progression of heart failure and further deterioration of patient health.

    Nurses’ care for such patients is more complex and involves timely implementation of therapy, daily physical examination, control of vital functions, keeping the balance of fluids, weight control, telemetric monitoring, psychological support. It is of utmost importance to recognize the changes in any of the mentioned segments so therefore enabling timely intervention and in doing so helping the patient to achieve the regression of hyperthyroidism and thus ‘stabilization’ of heart failure.

    Literature

    1. 1.
      Narayana SK, Woods DR, Boos CJ. Management of amiodarone-related thyroid problems. Ther Adv Endocrinol Metab. 2011;2(3):115–26.DOI
    2. 2.
      Harjai KJ, Licata AA. Amiodarone-induced hyperthyroidism: a case series and brief review of literature. Pacing Clin Electrophysiol. 1996;19(11 Pt 1):1548–54.DOI